Sometimes it works, sometimes it doesn't
I'm exporting from RefWorks into Zotero by exporting in EndNote/Procite. First try worked well, except it cut off after 15 of the references (with "check for error in file" message.) Next time, I did the same thing but got the "No translator" message. So far I've tried about 10 times, following the same steps each time, but have only had 3 successes. Any ideas?
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Thanks!
This one imported fine:
TY - JOUR
ID - 382
A1 - Reich,John W.
A1 - Johnson,Lisa M.
A1 - Zautra,Alex J.
A1 - Davis,Mary C.
T1 - Uncertainty of Illness Relationships with Mental Health and Coping Processes in Fibromyalgia Patients
Y1 - 2006
Y2 - Aug
VL - 29
IS - 4
SP - 307
AB - Fibromyalgia syndrome (FMS) is a chronic musculoskeletal pain condition poorly understood in terms of etiology and treatment by both physicians and patients. This condition of "uncertainty of illness" was examined as a variable involved in the adjustment of FMS patients, relating it to their depression, anxiety, affect, and coping styles. Fifty-one community-residing FMS patients provided self-report information on subsets of adjustment variables. Both cross-sectional and more dynamic longitudinal analyses showed that illness uncertainty was significantly associated with anxiety, negative affect, and avoidant and passive coping. Its positive relationship with depression was eliminated when a control variable, pain helplessness, was included as a covariate. Longitudinally, illness uncertainty interacted with interpersonally stressful daily events in predicting reports of reduced positive affect, suggesting that illness uncertainty acts as a risk factor for affective disturbances during stressful times. Implications of these results for therapeutic interventions are discussed.
A3 - Anonymous
SN - 01607715
JF - Journal of Behavioral Medicine
JA - Journal of Behavioral Medicine
UR - http://proquest.umi.com/pqdweb?did=1085046441&Fmt=7&clientId=65345&RQT=309&VName=PQD
M1 - Journal
ER -
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THIS ONE WOULDN'T IMPORT--ERROR MESSAGE:
TY - JOUR
ID - 383
A1 - Wood,Patrick B.
T1 - Fibromyalgia Syndrome: A Central Role for the Hippocampus--A Theoretical Construct
Y1 - 2004
VL - 12
IS - 1
SP - 19
EP - 26
KW - central nervous system
KW - fibromyalgia
KW - hyperactivity
KW - stress exposure
KW - hippocampus dysfunction
KW - glucocorticoid levels
KW - Glucocorticoids
KW - Hippocampus
KW - Hyperkinesis
KW - Muscular Disorders
KW - Stress
AB - Objective: A growing body of evidence implicates the central nervous system as playing a primary role in the diverse phenomena associated with fibromyalgia, including hyperactivity of stress systems and enhanced nociception. The objective of this review is to propose a unifying theory to explain a majority of these. Findings: Stress exposure causes deleterious changes within the central nervous system, the hippocampus being particularly vulnerable. The hippocampus is perhaps best known for its role in memory and cognition, two functions which are impacted by elevated glucocorticoid levels such as occur in prolonged stress. The hippocampus also provides inhibitory drive to brain centers associated with the stress response, i.e., the hypothalamic paraventricular nucleus, central amygdala, and locus coeruleus. In addition, the hippocampus has been demonstrated to participate in nociception, a function positively correlated with the activity of hippocampal N-methyl-D-aspartate [NMDA] subtype glutamate receptors. A variety of stress-related hormones are known to enhance the activity of hippocampal NMDA receptors, thereby increasing excitatory neurotransmission within the hippocampus. While the impact of stress-related hormones on hippocampal NMDA receptor function is adaptive in the acute scenario, exposure to chronic stress eventually leads to hippocampal dysfunction and atrophy secondary to excessive excitatory neurotransmission [i.e., excitotoxicity]. Conclusion: Fibromyalgia is characterized by abnormalities that appear to be related to hippocampal dysfunction, including hyperactivity of both corticotropin-releasing hormone neurons and the sympathetic nervous system, impaired declarative memory, and enhanced NMDA receptor- mediated nociception. It is therefore postulated that stress-induced, NMDA receptor-mediated dysfunction within the hippocampus plays a central role in the etiopathogenesis and clinical phenomena of fibromyalgia. (PsycINFO Database Record (c) 2007 APA, all rights reserved) (from the journal abstract)
N1 - 19177-002. First Author & Affiliation: Wood, Patrick B.; Department of Family Medicine, Louisiana State Health Science Center, Shreveport, LA, US. Other Journal Title: Journal of Musculoskeletal Pain. Release Date: 20041025. Publication Type: Journal (0100) Peer Reviewed Journal (0110). Media Covered: Print. Media Available: Electronic; Print. Document Type: Journal Article. Language: English. Major Descriptor: Central Nervous System; Glucocorticoids; Hippocampus; Hyperkinesis; Muscular Disorders. Minor Descriptor: Stress. Classification: Physical & Somatoform & Psychogenic Disorders (3290) . Population: Human (10) . Methodology: Literature Review. References Available: Y.
PB - Haworth Press
CY - US
A3 - Anonymous
SN - 1058-2452; 1540-7012
AD - Wood, Patrick B., Department of Family Medicine, Louisiana State Health Science Center, 1501 Kings Highway, Shreveport, LA, US, 71103, pwood@lsuhsc.edu
JF - Journal of Musculoskeletal Pain
JA - J.Musculoskeletal Pain
UR - pwood@lsuhsc.edu; http://search.ebscohost.com/login.aspx?direct=true&db=psyh&AN=2004-19177-002&site=ehost-live
M1 - Journal
ER -
Thanks again.
Lynn