Export issues from Embase (Ovid)
When using the Chrome extension to export references from Embase (Ovid) to Zotero (Windows Standalone), there are issues with the references when they appear in Zotero. For instance, the authors names are reverse (first name initial appears as last name and last name appears as first name). Then, for articles, the volume, issue and page numbers are all missing. Is there a way to look into these issues? Otherwise, the title, abstract, journal name, year and doi are all properly exported.
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VN - Ovid Technologies
DB - Embase
UI - 607059520
EU - 2015552884
PM - 26522738 [http://www.ncbi.nlm.nih.gov/pubmed/?term=26522738]
ST - EMBASE
AU - Sriram C.S.
AU - Jangra A.
AU - Gurjar S.S.
AU - Mohan P.
AU - Bezbaruah B.K.
AE - Bezbaruah B.K.; bez_bikki@yahoo.co.in
IN - (Sriram, Jangra, Bezbaruah) Department of Pharmacology and Toxicology, National Institute of Pharmaceutical Education and Research (NIPER), Guwahati, Assam 781032, India
(Gurjar) Department of Biotechnology, National Institute of Pharmaceutical Education and Research (NIPER), Guwahati, Assam 781032, India
(Bezbaruah) Department of Pharmacology, Gauhati Medical College, Guwahati, Assam 781032, India
(Mohan) Department of Pharmacology and Toxicology, College of Veterinary Science, Assam Agricultural University, Khanapara, Guwahati, Assam 781022, India
AD - B.K. Bezbaruah, Department of Pharmacology and Toxicology, National Institute of Pharmaceutical Education and Research (NIPER), Narkachal Hilltop, Bhangagarh, Guwahati, Assam 781032, India
CP - United States
TI - Edaravone abrogates LPS-induced behavioral anomalies, neuroinflammation and PARP-1.
SO - Physiology and Behavior. 154 (pp 135-144), 2016. Date of Publication: February 01, 2016.
PB - Elsevier Inc. (E-mail: usjcs@elsevier.com)
TJ - Physiology and Behavior
KW - Anxiety
KW - Depressive-like behavior
KW - Edaravone
KW - Lipopolysaccharide
KW - Poly(ADP-ribose) polymerase-1
UR - http://www.elsevier.com/locate/physbeh
MH - anhedonia
MH - animal experiment
MH - animal model
MH - animal tissue
MH - antidepressant activity
MH - antiinflammatory activity
MH - antioxidant activity
MH - *anxiety disorder/dt [Drug Therapy]
MH - anxiety disorder/dt [Drug Therapy]
MH - article
MH - behavior assessment
MH - behavior change
MH - biochemical analysis
MH - controlled study
MH - corticosterone blood level
MH - cytokine release
MH - *depression/dt [Drug Therapy]
MH - depression/dt [Drug Therapy]
MH - dose response
MH - drug effect
MH - drug mechanism
MH - elevated plus maze test
MH - enzyme linked immunosorbent assay
MH - exploratory behavior
MH - forced swim test
MH - hippocampus
MH - light dark test
MH - lipid peroxidation
MH - male
MH - mouse
MH - *nervous system inflammation/dt [Drug Therapy]
MH - nervous system inflammation/dt [Drug Therapy]
MH - nitrosative stress
MH - nonhuman
MH - open field test
MH - oxidative stress
MH - priority journal
MH - protein expression
MH - sucrose preference test
MH - tail suspension test
MH - tranquilizing activity
MH - Western blotting
MH - brain derived neurotrophic factor/ec [Endogenous Compound]
MH - catalase/ec [Endogenous Compound]
MH - corticosterone/ec [Endogenous Compound]
MH - Escherichia coli lipopolysaccharide
MH - glutathione/ec [Endogenous Compound]
MH - interleukin 1beta/ec [Endogenous Compound]
MH - malonaldehyde/ec [Endogenous Compound]
MH - nicotinamide adenine dinucleotide/ec [Endogenous Compound]
MH - *nicotinamide adenine dinucleotide adenosine diphosphate ribosyltransferase 1/ec [Endogenous Compound]
MH - nitrite/ec [Endogenous Compound]
MH - *norphenazone/do [Drug Dose]
MH - *norphenazone/dt [Drug Therapy]
MH - *norphenazone/ip [Intraperitoneal Drug Administration]
MH - *norphenazone/pd [Pharmacology]
MH - superoxide dismutase/ec [Endogenous Compound]
MH - tumor necrosis factor alpha/ec [Endogenous Compound]
MH - anxiety
MH - gene expression
MH - gene expression regulation
MH - gene inactivation
MH - gene overexpression
MH - *nervous system inflammation
MH - Swiss Webster mouse
MH - brain derived neurotrophic factor
MH - cytokine
MH - *lipopolysaccharide
MH - *nicotinamide adenine dinucleotide adenosine diphosphate ribosyltransferase 1
MH - *norphenazone
MH - scavenger
XT - anxiety disorder / drug therapy / norphenazone
XT - depression / drug therapy / norphenazone
XT - nervous system inflammation / drug therapy / norphenazone
XT - norphenazone / drug therapy / anxiety disorder
XT - norphenazone / drug therapy / depression
XT - norphenazone / drug therapy / nervous system inflammation
AB - Poly(ADP-ribose) polymerase-1 (PARP-1) is a DNA nick-sensor enzyme that functions at the center of cellular stress response and affects the immune system at several key points, and thus modulates inflammatory diseases. Our previous study demonstrated that lipopolysaccharide (LPS)-induced depressive-like behavior in mice can be ameliorated by 3-aminobenzamide, which is a PARP-1 inhibitor. In the present study we've examined the effect of a free radical scavenger, edaravone pretreatment against LPS-induced anxiety and depressive-like behavior as well as various hippocampal biochemical parameters including PARP-1. Male Swiss albino mice were treated with edaravone (3 & 10. mg/kg. i.p.) once daily for 14. days. On the 14th day 30. min after edaravone treatment mice were challenged with LPS (1. mg/kg. i.p.). After 3. h and 24. h of LPS administration we've tested mice for anxiety and depressive-like behaviors respectively. Western blotting analysis of PARP-1 in hippocampus was carried out after 12. h of LPS administration. Moreover, after 24. h of LPS administration serum corticosterone, hippocampal BDNF, oxido-nitrosative stress and pro-inflammatory cytokines were estimated by ELISA. Results showed that pretreatment of edaravone (10. mg/kg) ameliorates LPS-induced anxiety and depressive-like behavior. Western blotting analysis showed that LPS-induced anomalous expression of PARP-1 significantly reverses by the pretreatment of edaravone (10. mg/kg).Biochemical analyses revealed that LPS significantly diminishes BDNF, increases pro-inflammatory cytokines and oxido-nitrosative stress in the hippocampus. However, pretreatment with edaravone (10. mg/kg) prominently reversed all these biochemical alterations. Our study emphasized that edaravone pretreatment prevents LPS-induced anxiety and depressive-like behavior, mainly by impeding the inflammation, oxido-nitrosative stress and PARP-1 overexpression. Copyright © 2015.
RF - 80
EC - Clinical and Experimental Pharmacology [30], Psychiatry [32], Drug Literature Index [37], General Pathology and Pathological Anatomy [5], Neurology and Neurosurgery [8]
TN - Sigma Aldrich [United States]
RN - 218441-99-7 (brain derived neurotrophic factor); 9001-05-2 (catalase); 50-22-6 (corticosterone); 70-18-8 (glutathione); 542-78-9 (malonaldehyde); 53-84-9 (nicotinamide adenine dinucleotide); 14797-65-0 (nitrite); 89-25-8 (norphenazone); 37294-21-6 (superoxide dismutase); 9016-01-7 (superoxide dismutase); 9054-89-1 (superoxide dismutase)
EZ - EC 1.11.1.6 (catalase); EC 1.15.1.1 (superoxide dismutase).
IS - 0031-9384
EN - 1873-507X
DO - http://dx.doi.org/10.1016/j.physbeh.2015.10.029
CD - PHBHA
LG - English
SL - English
PT - Journal: Article
EM - 201641
DD - 20151224
DC - 20151209
YR - 2016
CR - Copyright 2015 Elsevier B.V., All rights reserved.
XL - http://ovidsp.ovid.com/ovidweb.cgi?T=JS&CSC=Y&NEWS=N&PAGE=fulltext&D=emedx&AN=607059520
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VN - Ovid Technologies
DB - Embase
UI - 612339389
EU - 20160699340
ST - EMBASE
AU - Smyth R.M.D.
AU - Taylor W.
AU - Heazell A.E.
AU - Furber C.
AU - Whitworth M.
AU - Lavender T.
AE - Smyth R.M.D.; rebecca.smyth@manchester.ac.uk
IN - (Smyth, Furber, Lavender) University of Manchester, Division of Nursing Midwifery and Social Work, Manchester M13 9PL, United Kingdom
(Taylor, Heazell, Whitworth) Maternal and Fetal Health Research Centre 5th Floor (Research), St Mary's Hospital Central Manchester NHS Foundation Trust, St Mary's Hospital, Manchester M13 9WL, United Kingdom
AD - R.M.D. Smyth, University of Manchester, Division of Nursing Midwifery and Social Work, Manchester M13 9PL, United Kingdom. E-mail: rebecca.smyth@manchester.ac.uk
CP - United Kingdom
TI - Women's and clinicians perspectives of presentation with reduced fetal movements: A qualitative study.
SO - BMC Pregnancy and Childbirth. 16 (1) (no pagination), 2016. Article Number: 280. Date of Publication: 26 Sep 2016.
PB - BioMed Central Ltd. (E-mail: info@biomedcentral.com)
TJ - BMC Pregnancy and Childbirth
KW - Clinicians
KW - Interviews
KW - Management
KW - Maternal experience
KW - Qualitative
KW - Reduced fetal movements
UR - http://www.biomedcentral.com/bmcpregnancychildbirth/
MH - adverse outcome
MH - anxiety
MH - clinical article
MH - data analysis
MH - doctor patient relation
MH - family
MH - female
MH - *fetus movement
MH - human
MH - maternal care
MH - pregnancy
MH - pregnant woman
MH - *qualitative research
MH - *semi structured interview
MH - social network
AB - Background: Worldwide maternal perception of fetal movements has been used for many years to evaluate fetal wellbeing. It is intuitively regarded as an expression of fetal well-being as pregnancies in which women consistently report regular fetal movements have very low morbidity and mortality. Conversely, maternal perception of reduced fetal movements is associated with adverse pregnancy outcomes. We sought to gain insight into pregnant women's and clinicians views and experiences of reduced movements. Method: We performed qualitative semi-structured interviews with pregnant women who experienced reduced fetal movements in their current pregnancy and health professionals who provide maternity care. Our aim was to develop a better understanding of events, facilitators and barriers to presentation with reduced fetal movements. Data analysis was conducted using framework analysis principles. Results: Twenty-one women and 10 clinicians were interviewed. The themes that emerged following the final coding were influences of social network, facilitators and barriers to presentation and the desire for normality. Conclusions: This study aids understanding about why women present with reduced movements and how they reach the decision to attend hospital. This should inform professionals' views and practice, such that appreciating and addressing women's concerns may reduce anxiety and make presentation with further reduced movements more likely, which is desirable as this group is at increased risk of adverse outcome. To address problems with information about normal and abnormal fetal movements, high-quality information is needed that is accessible to women and their families. Copyright © 2016 The Author(s).
RF - 32
EN - 1471-2393
DO - http://dx.doi.org/10.1186/s12884-016-1074-x
CD - BPCMB
LG - English
SL - English
PT - Journal: Article
EM - 201641
DD - 20161004
DC - 20161004
YR - 2016
CR - Copyright 2016 Elsevier B.V., All rights reserved.
XL - http://ovidsp.ovid.com/ovidweb.cgi?T=JS&CSC=Y&NEWS=N&PAGE=fulltext&D=emedx&AN=612339389
As you can probably imagine, getting the page, volume and issue information from the way it's displayed in a citation (which isn't always formatted the same way) is very tricky (they each should have their own tag), so not sure how much we can do, but I'll certainly try.
It's really a shame that OVID doesn't invest more in better metadata.
Those articles are in PubMed with public domain, super-high-quality metadata, so it's really hard to understand.
...really a shame that OVID doesn't invest...
It was a few years ago that I talked with an OVID representative on behalf of a student and later in another context (I had to conversationally convince someone there that I wasn't capturing their metadata for use in SafetyLit after written correspondence didn't make sense) about their inconsistent, incoherent, often incorrect, and strange formatting of metadata. The OVID representative made it clear that their garbled metadata was intentional. It is to prevent students from "willy willy nilly nilly" capturing their protected catalog to "make a gigantically massive" collection in violation of "perfectly, reasonably, proper" contract terms with the university.
I was so impressed with the rep's hyperbolic prose that I took note and sometimes use his words when needed for snark.
So it isn't that OVID doesn't invest to improve metadata, the opposite (investing effort to manipulate their metadata) appears to be the case.
Web of Science, Knowledge, etc. does something similar but with articles' titles. It adds hyphens and dashes where there are none and removing such when punctuation exists, interchanging articles (a, the), and performing other tricks. All this in an effort to protect their prouduct by inserting relatively benign Mountweazel copyright traps.
No other bibliographic management software can do a better job of importing Ovid metadata.
If the reference librarian doesn't seem to understand your problem, ask if there is someone on the staff who does trainings on the use of personal bibliography managers such as EndNote, Mendeley, or Zotero. That person _will_ understand and may be able to help get Ovid's attention. I hope that if enough libraries complain Ovid will take action.
The last time we tested EndNote on Ovid data it was actually quite bad. I don't have access anymore, so hard for me to tell anymore.